A B C D E F G H I K L M N O P R S T U V W X Z
WNK1 (WNK lysine deficient protein kinase 1)
Delaloy C, Hadchouel J, Imbert-Teboul M, Clemessy M, Houot AM, Jeunemaitre X. Cardiovascular expression of the mouse WNK1 gene during development and adulthood revealed by a BAC reporter assay. Am J Pathol 2006; 169:105-18.
Garcia EA, Newhouse S, Caulfield MJ, Munroe PB. Genes and hypertension. Curr Pharm Des 2003; 9:1679-89.
Katsuya T, Ogihara T. Trends in hypertension research. Nippon Rinsho 2006; 64 Suppl 5:5-11.
Lafreniere RG, MacDonald ML, Dube MP et al. Identification of a novel gene (HSN2) causing hereditary sensory and autonomic neuropathy type II through the Study of Canadian Genetic Isolates. Am J Hum Genet 2004; 74:1064-73.
Mein CA, Caulfield MJ, Dobson RJ, Munroe PB. Genetics of essential hypertension. Hum Mol Genet 2004; 13:169-75.
Newhouse S, Farrall M, Wallace C et al. Polymorphisms in the WNK1 gene are associated with blood pressure variation and urinary potassium excretion. PLoS One 2009. doi:10. 1371/journal. pone. 0005003.
Osada Y, Miyauchi R, Goda T et al. Variations in the WNK1 gene modulates the effect of dietary intake of sodium and potassium on blood pressure determination. J Hum Genet 2009; 54:474-8.
Roddier K, Thomas T, Marleau G et al. Two mutations in the HSN2 gene explain the high prevalence of HSAN2 in French Canadians. Neurology 2005; 64:1762-7.
Tobin MD, Timpson NJ, Wain LV et al. Common variation in the WNK1 gene and blood pressure in childhood: the Avon longitudinal study of parents and children. Hypertension 2008; 52:974-9.
Turner ST, Schwartz GL, Chapman AB, Boerwinkle E. WNK1 kinase polymorphism and blood pressure response to a thiazide diuretic. Hypertension 2005; 46:758-65.
Xie J, Wu T, Xu K, Huang IK, Cleaver O, Huang CL. Endothelial-specific expression of WNK1 kinase is essential for angiogenesis and heart development in mice. Am J Pathol 2009; 175:1315-27.
Zambrowicz BP, Abuin A, Ramirez-Solis R et al. Wnk1 kinase deficiency lowers blood pressure in mice: a gene-trap screen to identify potential targets for therapeutic intervention. Proc Natl Acad Sci USA 2003; 100:14109-14.
WNT5B (Wingless-type MMTV integration site family, member 5B)
Huang RS, Duan S, Bleibel WK et al. A genome-wide approach to identify genetic variants that contribute to etoposide-induced cytotoxicity. Proc Natl Acad Sci USA 2007; 104:9758-63.
Salpea KD, Gable DR, Cooper JA et al. The effect of WNT5B IVS3C>G on the susceptibility to type 2 diabetes in UK Caucasian subjects. Nutr Metab Cardiovasc Dis 2009; 19:140-5.
